Notch signaling mediated by TGF-β/Smad pathway in concanavalin A-induced liver fibrosis in rats

نویسندگان

  • Yi Wang
  • Ruo-Wu Shen
  • Bing Han
  • Zhen Li
  • Le Xiong
  • Feng-Yu Zhang
  • Bei-Bei Cong
  • Bei Zhang
چکیده

AIM To explore the exact interaction between Notch and transforming growth factor (TGF)-β signaling in liver fibrosis. METHODS We established a rat model of liver fibrosis induced by concanavalin A. Peripheral blood mononuclear cells (PBMCs) were isolated from the modeled rats, and cultured with γ-secretase inhibitor DAPT and TGF-β inhibitor for 24 h. The mRNA levels of Notch and TGF-β signaling were detected by quantitative real-time polymerase chain reaction. Expression of Notch and TGF-β proteins was analyzed by western blotting. RESULTS Compared to control rats, Notch and TGF-β signaling was activated in PBMCs of model rats. Administration of DAPT and TGF-β inhibitor suppressed Notch and TGF-β signal transducer in PBMCs of model rats. DAPT reduced mRNA and protein expression of TGF-β signaling, such as TGF-β1 and Smad3. TGF-β inhibitor also downregulated Notch1, Hes1 and Hes5, and mRNA and protein expression of the Notch signaling pathway. CONCLUSION Notch and TGF-β signaling play a role in liver fibrosis. TGF-β signaling upregulates Notch signaling, which promotes TGF-β signaling.

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عنوان ژورنال:

دوره 23  شماره 

صفحات  -

تاریخ انتشار 2017